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The dysfunction of BNC1 activates ferroptosis through the Nrf2-Hippo pathway in oocytes, resulting in POI (44)
Dong YH, Wu JH, Chang CH, et al
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Moderately and severely affected individuals have enzymes with dysfunctional catalytic sites, rendering it unable to participate in detoxification reactions
Despite these limitations, ongoing investigation is expected to yield deeper insights into the mitochondrial pathophysiology of AKI and support the development of more effective and clinically translatable therapeutic strategies
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